Neuro-Mito Repair 

Description

The Neuro-Mito Repair Research Kit combines NAD⁺ (500mg) and MOTS-c (40mg) — targeting the NAD⁺ metabolism and mitochondria-to-nucleus retrograde signaling pathways within a focused two-component neural and mitochondrial repair research panel.

Kit Components

• NAD⁺ (500mg): Sirtuin (SIRT1–7) substrate and PARP cofactor central to mitochondrial biogenesis, DNA repair, and metabolic gene expression. NAD⁺ levels decline in neural tissue with age, driving mitochondrial dysfunction in neurodegenerative models.
• MOTS-c (40mg): Mitochondria-derived 16-AA peptide that activates AMPK and nuclear Nrf2/ARE programs. In neural tissue models, MOTS-c has been investigated for protection against oxidative stress and metabolic challenge.

Research Context

Mitochondrial dysfunction is central to neurodegeneration — and both components address it from different directions. NAD⁺ restores sirtuin activity and supports mitochondrial biogenesis via PGC-1α. MOTS-c activates AMPK and Nrf2 antioxidant programs that protect mitochondria from ROS damage. Studying them together provides a comprehensive view of NAD⁺-AMPK-mitochondria signaling in neural models.

Common Research Applications

• Neural mitochondrial function and NAD⁺ restoration studies
• AMPK-sirtuin pathway interaction in neural tissue
• Neurodegeneration model mitochondrial protection research
• Nrf2 antioxidant pathway and oxidative stress studies

For Research Use Only. Not for human or veterinary use.